(PPF) inactivates broad spectrum of virusesCinnovir
Pre-clinical studies have determined that PPF inactivates:
- Orthomyxoviridea – Influenza types H1N1, H9N2, H5N1, H5N3
- Paramyxoviridea – Parainfluenza, Newcastle disease
- Herpesviridea – Herpes Simplex 1, Herpes Simplex 2
- Retroviridea – HIV
- SARS-CoV-2
Safety
- is a natural water extract derived from cinnamon cassia (Cinnamomum cassia) solely by physical separation, utilizing no chemicals or solvents.Cinnovir
- Ground cinnamon is GRAS-confirmed and has been used for centuries in food preparation. ()https://reference.medscape.com/drug/cinnamon-batavia-cassia-344478
- 300 mg of is derived from 10 grams of ground cinnamon.Cinnovir
- There is some concern related to high intake of due to one of its constituents, coumarin, which if consumed in high doses can cause liver damage.Cinnamomum cassia
- This concern is irrelevant in our case since Novid-20 products are virtually coumarin-free.
SARS-CoV-2 Study
- The binding capacity of the lentivirus is about 200 times greater than that of SARS-CoV-2.
- The study found that rates of infection with the lentivirus decreased by approximately 60% after treatment with PPF, indicating that PPF may be effective in blocking SARS-CoV-2 and potentially other viruses as well. A lab study has been conducted (2021, Hebrew University) on PPF, examining potential antiviral activity of PPF by changes of the Spike-hACE2 interactions, using Spike-pseudotyped lentivirus.
- Additional data from this study is available upon request.
Anti-Viral Activity of Cinnovir Against HSV-1
Pretreatment with Cinnovir Gel mitigated HSV-1 induced cytotoxicity in VERO E6 in a dose-dependent manner. A concentration of 50ug/ml significantly decreased the viral cytopathic effects. The anti-viral activity peaked at a concentration of 75ug/ml with similar activity observed at higher concentrations.
References will be provided upon request.
Pre-clinical Proof of Concept – HSV-2
Dose-dependent protection by Cinnovir against viral induced cytotoxicity in VERO E6 Cells 48 h post infection with HSV-2.
